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Humanin is a naturally occurring micro-peptide found in mitochondria. It protects cells from apoptosis and may be useful in staving off heart, eye, brain, and muscle diseases. Humanin is one of a select, small class of compounds targeting mitochondrial function and thus improving cellular energy efficiency and homeostasis is a molecule consisting of a lipid connected to a peptide. Humanin decreases age-related cognitive decline in mice
To examine if humanin may improve cognition in a mammalian model of aging, 18-month-old, female C57Bl/6N mice were obtained from the National Institute on Aging (NIA) and treated with HNG biweekly by IP injection at a dose of 4 mg/kg. This dose was chosen as it was shown to have physiological effects17. At 24-months of age (6 months of treatment) (N = 24 and 23 mice for control and HNG groups respectively), the HNG-treated group maintained their balance longer than the control group on a rotarod test (Fig. 2A). This result was not due to differences in body weight as there was not a significant difference between the two groups of mice selected for this test (data not shown). Four months later, the trend was the same but the difference between the two groups was smaller and became non-significant. RELATED PRODUCTS
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Aging is associated with a number of biological changes including decreases in memory and cognitive function. Humanin is the first member of a new class of peptides originating from small, alternative open reading frames within the mitochondrial genome. Since it was discovered, humanin has been shown to be neuroprotective in multiple in vitro and animal studies1. The importance of the mitochondria in the etiology of Alzheimer’s disease (AD) is becoming more apparent and evidence suggests that humanin protects from various insults both in cellular models and in vivo models of Alzheimer’s disease2–5. In vitro, humanin protects against amyloid-beta (Aβ) toxicity as well as other familial forms of AD. In vivo rodent studies have shown that humanin also prevents the memory deficits caused by intracerebroventricular injection of Aß25–35 as well as having potent protective properties in the triple-transgenic AD mouse5,6. Thus, humanin is a neuroprotective factor whose endogenous production could influence AD progression and other neurodegenerative diseases. MORE ON PEPTIDES
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