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Buy AICAR 50mg Online is an analogue of adenosine monophosphate. It has been used clinically to protect against cardiac ischemia following heart attack. AICAR has antioxidant properties and may therefore help to slow the physiologic signs of aging. There is ongoing research into the use of AICAR to mediate the effects of diabetes, auto-immune diseases, and other inflammatory conditions.

AICAR CHEMICAL DEFINITION

AICAR (5-aminoimidazole-4-carboxamide-1-β-D-ribofuranoside; or often also called as AICA ribonucleotide) is an synthetic analog of adenosine monophosphate (AMP), that stimulates AMP-dependent protein kinase (AMPK) activity. AICAR has chemical formula C9H15N4O8P and molar mass 338.213 g·mol−1. The AMPK-stimulating AICAR is synthesized in a lab and is being evaluated in preclinical research and human clinical trials as a therapeutic agent to treat certain metabolic disorders in humans.

ADENOSINE MONOPHOSPHATE AND AMP ACTIVATED PROTEIN KINASE

Nucleotides are molecules consisting of a nucleoside and a phosphate group, they are the basic building blocks of DNA and RNA, and also play a central role in metabolism at a fundamental, cellular level. Nucleotide Adenosine monophosphate (AMP; or also known as 5′-adenylic acid) is a substance produced naturally by the body, that consists of a phosphate group, the sugar ribose, and the nucleobase adenine; it is an ester of phosphoric acid and the nucleoside adenosine. Adenosine monophosphate plays an important role in many cellular metabolic processes, being interconverted to ADP and/or ATP. AMP is also a component in the synthesis of RNA

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AMP activated protein kinase (AMPK; 5′ adenosine monophosphate-activated protein kinase) is an enzyme and protein, that regulates metabolism in a variety of ways. AMPK acts as an energy regulator and is activated during exercise or other circumstances that use up cellular energy. It plays a role in cellular energy homeostasis, largely to activate glucose and fatty acid uptake and oxidation when cellular energy is low. AMPK is expressed in a number of tissues, including the liver, brain, and skeletal muscle. In response to binding AMP and ADP, the net effect of AMPK activation is stimulation of hepatic fatty acid oxidation, ketogenesis, stimulation of skeletal muscle fatty acid oxidation and glucose uptake, inhibition of cholesterol synthesis, lipogenesis, and triglyceride synthesis, inhibition of adipocyte lipogenesis, inhibition of adipocyte lipolysis, and modulation of insulin secretion by pancreatic beta-cells.